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Experimental work examining angiogenic responses during muscle and tendon healing reported increased expression of angiogenesis-associated markers such as vascular endothelial growth factor (VEGF), CD34, and factor VIII within injured tissues following BPC-157 exposure [6]
Likewise, there is greater proliferation of human umbilical vein endothelial cells (HUVECs), significantly promoted migration of HUVECs (transwell assay), and the upregulated expression of VEGF-a, and accelerated vascular tube formation in vitro (Huang et al., 2015)
In pre-clinical research, inhibiting NNMT is studied for its effects on NAD+ salvage pathway activity and methyl-donor availability
The clinical version is narrower, better defined, and more reliably delivered
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